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Acad Modern Digestion & Intervention "-2001 San Jiu Wei Tai on rats before and after treatment of acute gastritis Histopathology of
San Jiu Wei Tai on rats before and after treatment of acute gastritis Histopathology ofYou can in the "My Services" in the you add a reference to the notification list, and configure access to notices.San Jiu Wei Tai has anti-inflammatory analgesic, Qi Jianwei effect, treating superficial gastritis, erosive gastritis and atrophic gastritis. This through the establishment of experimental rat model of acute gastritis, before and after their treatment of pathological changes in gastric mucosa study to study in the organization for the experimental treatment of acute gastritis corroboration; Hp through its inhibition in vitro experiments to the treatment of chronic gastritis provide experimental evidence for the mechanism. of: Zhang Min car bore the ancient Qiang Yi Ya Zhong Chunhua Zhang Min, Li Qiang, pore old Ya, Zhong Chunhua (610041, Chengdu Huaxi Medical University, Center for Drug Preclinical pharmacology)
Car Art (400000, Chongqing, Department of Pathophysiology, Chongqing Medical College) Title: Modern Digestion & Intervention ISTIC English title: MODERN DIGESTION
Pathophysiology of acute gastritis 10:18
Changes in the pathophysiology of acute gastritis are as follows:
Lesions can be diffuse or limited to the gastric mucosa of catarrhal inflammation. Mucosal congestion and edema, exudate and mucus surface coverag


e can be a bit like bleeding and different degrees of erosion. Membrane due to a lymphocyte, neutrophils, plasma cells and a few eosinophils infiltration, edema, mucosal vascular congestion, occasional small interstitial hemorrhage, severe submucosal edema, congestion on Collecting medical `Education Network .Close News How to Learn the pathophysiology pathophysiology nature of the subject, the task of hemorrhagic gastritis (pictures) were happy to stomach (acid and anti-ulcer drugs) chronic atrophic gastritis pathological Livzon triple (acid and anti-ulcer drug) Armillarisin (hepatobiliary disease adjuvant) consciousness and the pathophysiology of Hp gastritis in the stomach Kang Su (Zhuxiaohuayao) Synthetic aluminum silicate (acid and anti-ulcer drug) Pathophysiology compound allantoin (anti-acid and anti-ulcer drugs) proglumide (acid and anti-ulcer drug) An Weiling (gastrointestinal spasmolytic) Pathophysiology - Pathophysiology normal serum sodium - pathophysiology of hypernatremia - hyponatremia pathophysiology - the regulation of body fluid volume and pathophysiology of osmotic pressure - fluid and electrolyte composition distribution
Acute gastroenteritis is the intestinal dysentery, cholera, typhoid, a variety of other pathogens (including bacteria and viruses) caused by acute gastrointestinal infections. Occur in summer and autumn, often through unclean food and water, by mouth intrusion. Acute gastroenteritis is a disease causes a variety of reasons, such as bacteria Kang Jiajian Recommended ing
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Acute gastroenteritis is the intestinal dysentery, cholera, typhoid, a variety of other pathogens (including bacteria and viruses) caused by acute gastrointestinal infections. Occur in summer and autumn, often through unclean food and water, by mouth intrusion.
Cause disease
Acute gastroenteritis is a variety of reasons, such as bacteria, viruses, infections, toxins, chemicals, the role of the gastrointestinal tract caused by acute, diffuse inflammation. Most of the ingestion of food with bacteria or toxins, such as bad, corrupt, contaminated staple foods caused. Occurred in summer and fall. Rapid onset of acute gastroenteritis, often within 24 hours of onset. Can be divided into acute gastritis, acute colitis, acute gastroenteritis III. By the chemical, physical (mechanical and temperature factors), such as microbial infection or bacterial toxins cause, the latter is more common. By microorganisms in the consumption of contaminated food and bacterial toxin-induced acute gastritis in simple, microbial, including Salmonella, halophilic bacteria, Helicobacter pylori, rotavirus and Norwalk virus, bacterial toxins Staphylococcus aureus toxin is more common. Pathophysiology
Chemicals, such as aspirin and other non-steroidal anti-inflammatory drugs inhibit cell mitochondria oxidative phosphorylation, thereby inhibiting the cell membrane Na-K-ATP enzyme and active transport system, leading to increased permeability of mucosal cells the Shuinazhuliu, cell swelling and loss; epoxide by inhibiting the enzyme can block the endogenous prostaglandin E2 and I2 synthesis, epithelial sodium bicarbonate and mucus secretion decreased, H anti-diffusion, thus undermining the gastric mucosa barrier.
Key words: Key words: acute gastroenteritis diet prostate Kangjia health network, access to health knowledge base, displaying the brand image of the platform


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