Health Worlds

LOS ANGELES, Oct. 21 Xinhua U.S. researchers have completed animal study found that rats, after suffering from Alzheimer's disease, the retina will be similar brain lesions. This means that, through the eye might be able to more easily detect early diagnosis and treatment of Alzheimer's disease.
Alzheimer's disease is more common in Alzheimer's disease, degenerative brain disease that the clinical symptoms of cognitive, memory and language barriers and other features appear. The medical profession now believe that Alzheimer's disease brain may be abnormal accumulation of amyloid caused.
University of California, Irvine, 21, issued a press announcement that the school researchers used genetic engineering to make mice suffering from Alzheimer's disease. The sick mice were observed and detected the system, they also appeared in the retina of the abnormal accumulation of amyloid.
In addition, the researchers also found that when the implementation of some of these sick mice experimental therapy, by observing the retina through the brain tissue than the observed effect is more convenient, more accurate and better able to predict the treatment of Alzheimer's s disease is effective.
Press release said: "the eyes are windows of the soul, and this research shows that the eyes may also be like a mirror reflecting the brain in Alzheimer's disease."
The researchers said that the medical profession is now being tested using brain imaging techniques in early diagnosi


s of Alzheimer's disease. Next consider the development of new retinal imaging techniques, diagnosis and treatment of Alzheimer's disease. In comparison, the retinal image on the human body less invasive, lower cost and easier implementation.

Abstract: Objective: Chrysophanol on -amyloid protein (A 25-35) caused by Alzheimer's disease (Alzheimer's disease, AD) mice with learning and memory impairment and brain glutamate content. Methods: Intracerebroventricular injection of mice with a one-time condensed A 25-353 g (1.0 mmol.L ^ -1) made AD model, intraperitoneal injection after model chrysophanol or solvent, continuous injection of 14d. Determined by passive avoidance learning and memory in mice, Brain and Determination of glutamate. Results: Compared with model group, phenol group into the chamber rhubarb latency, reducing the number of errors; mouse brain glutamate levels decrease. Conclusion: Chrysophanol on A 25-35 induced memory impairment in AD model mice has a protective effect and reduces glutamate levels in the brain. Number of pages: 3 pages :1-3 Keywords: learning and memory in glutamate chrysophanol A 25-35 subject categories: R9-33 [medicine, HEALTH "pharmacy] Related Articles: Topics related references (8)
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