Health Worlds

Lactic acidosis should be treated?
This disease is very important to prevent the following points should be noted:
1 liver and kidney dysfunction who were hanged there pyelonephritis biguanide diabetic nephropathy such as glomerular sclerosis, can cause build-biguanide; coronary heart disease associated with recessive renal circulation disorders such as heart failure may also affect the time excretion accompanied by hypoxia and therefore be required before use of biguanide drugs such as metformin liver and kidney function to identify the heart of the disease occurs more often than phenformin (phenformin) was significantly less
2 Where the shock when the lack of O2 liver and kidney failure if acidosis must be alert to the possibility of the disease and prevention efforts
A type of group treatment for primary disease, and shock should be corrected for the lack of O2-based treatment for B-group are as follows:
1 Disable the disease induced by drugs such as paracetamol and other phenformin various alcohols
2 correct the acidosis and electrolyte metabolism and water after diagnosis in the past by the emphasis in recent years, immediately to the large number of clinical and experimental proof of NaHCO3 in 24 hours early as a NaHCO3 <500mmol when only ineffective and can lead to pulmonary edema, hypotension, coma and lack of O2 increased illness see higher rates of anti-blood lactate (lack of O2) so Kassier other propositions to the small dose of NaHCO3 increas


ed HCO-3 to 4 ~ 6mmol / L and maintained at 14 ~ 16mmol / L aly, especially severe acidosis (pH <7.0) were lung function and should not be too quick to correct unless the cycle is very difficult to rule out CO2 as well, otherwise the result of lack of O2
The rest of intravenous therapy with glucose and insulin on diabetic ketoacidosis compared to those with significant
Mortality of the disease difficult to treat up to 50% or more should be emphasis on prevention
Article ID :1009 -5519 (2008) 04-0550-02 CLC number: R5 Document code: B
Pulmonary Encephalopathy (lung brain) is a chronic pulmonary heart disease is one of serious complications. Has confirmed that respiratory acidosis (respiratory acidosis) in the brain in the pathogenesis of the lung is important, but metabolic acidosis (acidosis) reported rarely. In this paper, on behalf of the acid in the brain occur in lung function and prognosis, treatment for a study.
1 Materials and Methods
1.1 Clinical data: 98 cases of chronic pulmonary heart disease in patients with acute exacerbation of the country in 1977 are in line with the diagnostic criteria for pulmonary heart disease, 50 cases of pulmonary meningitis group also comply with the 1980 Huangshan meeting diagnostic criteria for brain development of the lung. Lung brain age group (63.2 9.4) years, 26 males and 24 females, admitted the performance of the lung were 36 cases of brain after admission occurred during the treatment of lung cerebral symptoms in 14 cases. 20 cases of hospital mortality, mortality 40%. Non-lung average brain, control group (62.4 8.6) years. 27 males and 21 females. 5 patients died in hospital mortality 10%.
1.2 Methods: Blood gas values from the United States IL-BO2 blood gas analyzer automatically print the report. Serum electrolytes were determined by ion selective electrode method. Detection of blood in patients with femoral artery access the value of simultaneous determination of the venous serum K, Na, Cl-a total of 130 episodes. Brain detected 50 cases of 64 cases of lung times, in which the lung after brain detected 33 cases of hospital performance times. The course of treatment, pulmonary symptoms within 24 hours of testing brain 13 cases, pulmonary brain death within 24 hours before the test in 18 cases.
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