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Hypertrophic obstructive cardiomyopathy symptoms
Time: February 1, 2008 Font Size: [] online consultation online registration Abstract: onset and more slowly. About 1 / 3 have a family history. Most symptoms began 30 years ago. Men and women alike suffer. The main symptoms are: breathing difficulties, and more after the exertion is due to reduced left ventricular compliance, diastolic pressure is increased, followed by pulmonary vein pressure and pulmonary bleeding with the rules. With ventricular septal hypertrophy with mitral regurgitation can be maintained by increased pulmonary blood stasis. precordial pain, mostly in
Onset and more slowly. About 1 / 3 have a family history. Most symptoms began 30 years ago. Men and women alike suffer.
The main symptoms are: breathing difficulties, and more after the exertion is due to reduced left ventricular compliance, diastolic pressure is increased, followed by pulmonary vein pressure and pulmonary bleeding with the rules. With ventricular septal hypertrophy with mitral regurgitation can be maintained by increased pulmonary blood stasis. precordial pain, and more after the exertion, like angina, but may from typical aerobic myocardial hypertrophy due to increased coronary blood supply is relatively insufficient. fatigue, dizziness and syncope, occurred more in the activity is due to rapid heart rate, so that the aly poor diastolic left ventricular diastolic filling to further reduce, increase less than filling,


cardiac output decreased. Activity or emotional effect when the sympathetic contraction of the cardiac hypertrophy strengthened and increased outflow tract obstruction, cardiac output plummeted and cause symptoms. palpitations, due to cardiac dysfunction or arrhythmias due. heart failure, more common in patients with advanced, due to reduced myocardial compliance and ventricular end-diastolic pressure was significantly increased, and then atrial pressure is increased, and is often associated with atrial fibrillation. Extensive myocardial fibrosis in patients with advanced, ventricular systolic function is also weakened, prone to heart failure and sudden death.
Common signs are: heart dullness to the left to expand. Under the shift to the left apex beat, a lifting of the impulses. Or double apex beat, this is the heart to reduce ventricular ejection compliance generated when the pulse is reached before the apex beat. apical lower left sternal border can be heard inside the mid or late systolic ejection murmur, and not to the heart to the apex of the spmay be associated with systolic tremor, seen in patients with ventricular outflow obstruction. Where the increase in myocardial contractility or reduce cardiac measures such as load to digitalis, isoproterenol (2 g/min), amyl nitrite, nitroglycerin, for the Val-salva action, manual labor or premature beat after the post- can make noise enhancement; where weakened myocardial contractile force or increase the heart load measures, such as a vasoconstrictor, blockers, squat, fist clenched to make noise when you can be weakened. About half of the patients at the same time hear the murmur of mitral regurgitation. unusual split second sound can be presented, spurting blocked due to left ventricular and aortic valve closure caused delays. Associated with the third tone is common in patients with mitral regurgitation.
ECG: ST-T changes seen in more than 80% of patients, most of the normal coronary artery, a few limitations to the apex in patients with cardiac hypertrophy and the abnormal coronary artery has a huge inverted T wave. signs of left ventricular hypertrophy seen in 60% of patients, the existence and extent of cardiac hypertrophy and location related. the presence of abnormal Q waves. V6, V5, aVL, I lead a deep rather than wide on the Q wave, reflecting the asymmetric septal hypertrophy, do not be mistaken for myocardial infarction. Sometimes , , aVF, V1, V2 can also lead a Q wave, its occurrence may be related to left ventricular wall after the subendocardial myocardium and impulse conduction due to irregular and delayed. left atrial waveform abnormalities that may found in 1 / 4 patients. some patients with WPW syndrome.
Echocardiography: asymmetric septal hypertrophy, septal thickness and left ventricular posterior wall thickness ratio greater than 1.3:1, more emphasis on the levy in the past, but now found also found in other diseases such as hypertension, aortic stenosis. Two-dimensional method to measure the degree of left ventricular thickness is more useful. mitral valve in the systolic forward. reduced left ventricular cavity and outflow tract obstruction. left ventricular diastolic dysfunction, including reduced compliance, prolonged rapid filling, isovolumic relaxation time prolonged. Doppler method can be used to understand the origin of noise and calculated the pressure difference before and after obstruction.
Normal chest X ray may see increased left ventricle may also be in the normal range. X ray or radionuclide angiocardiography can show septal thickening, reduced left ventricular cavity. Radionuclide myocardial scan can show the location and extent of cardiac hypertrophy.
Cardiac catheterization showed elevated left ventricular end-diastolic pressure. Left ventricular outflow tract obstruction in the ventricular cavity and outflow tract systolic pressure difference between.
(Editor: great minds Network)
Key words: hypertrophic obstructive hypertrophic cardiomyopathy 7 pages: Previous 1 [2] 34567 Next


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