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Department OverIntroduction of different causes sustained increases in blood lactic acid and PH decreased (7.35 for a treatment. Should be prevention, timely detection and treatment, if necessary oxygen. II. Fluid expansion can improve tissue perfusion, correct shock, diuretic row acid. With saline, to avoid the use of preparations containing lactic acid. III. Make base: 5% sodium bicarbonate 100-200mlIV, when 7.25 when the stop input base, in order to avoid alkalosis. IV. Insulin: Insulin adding glucose infusion to reduce the sugar in the anaerobic glycolysis, which will help the elimination of lactic acid. Five. Hemodialysis: Shuinazhuliu commonly used in patients can not tolerate.
Lactic acid is a glycolytic intermediate metabolites. Under normal circumstances, lactate 50% to 60% in the liver into glycogen storage, about 30% to 35% of kidney use. When the blood lactate increased more than 2mmol / L, blood pH less than 7.35, the absence of any other cause acidosis can be diagnosed with lactic acidosis. High mortality rate of this disease (50%), is one of the acute complications of diabetes. Common causes can be divided into two main categories:
(1) no hypoxia and shock caused by the state. Found: drugs. Biguanide hypoglycemic agents, especially phenformin causes were more common and severe. Also: ethanol, methanol, xylitol, sorbitol, fructose, paracetamol, salicylates, streptozotocin, catecholamines, cyanide type, isoniazid, ethylene glycol can cause. systemic


disease. Diabetic ketoacidosis may be associated with this disease, liver disease, kidney failure uremia, cancer, leukemia, severe infections with sepsis, convulsions, anemia, hunger can cause the disease. genetic disease. Glucose -6- phosphate dehydrogenase deficiency, fructose 1,6 - dioxane acid enzyme deficiency, pyruvate carboxylase deficiency, pyruvate dehydrogenase deficiency, oxidative phosphorylation defects may also cause the disease.
(2) and the lack of shock caused by O.2. Such as: shock. As myocardial infarction, heart failure, severe trauma, infection, bleeding caused by cardiac, infectious, hemorrhagic shock dehydration. oxygen suffocation. Carbon monoxide poisoning, pulmonary embolism and infarction. acute pancreatitis with shock.
As a high mortality rate of this disease, it is necessary to strengthen prevention measures taken are as follows: Where a liver and kidney dysfunction, not the best biguanide hypoglycemic agents, diabetes, heart failure-prone heart disease, kidney circulation disorder may also affect the biguanide excretion of drugs, it should be used with caution; avoid the use of ethanol, methanol, xylitol, salicylates, isoniazid and other drugs, propranolol and other drugs used with caution; far as possible without fructose, sorbitol and the use of glucose, in order to avoid the disease; where shock, hypoxia, liver and kidney failure state of acidosis, should be to correct hypoxia, ischemia, correct shock as the basic measures to prevent the occurrence of this disease.

Location: Endocrino Diabetes lactic acid text diabetic lactic acidosis in the differential diagnosis in this select font: s: of: TCM doctor network : diabetic lactic acidosis and the diseases easily confused? the differential diagnosis of diabetic coma diabetic coma seizure medication history a history of low blood sugar body syndrome laboratory sudden sweating, palpitation, character abnormalities, coma, insulin, glibenclamide and other dilated pupils, heart rate fast, the
Diabetes, lactic acidosis and the diseases easily confused?
Differential diagnosis of diabetic coma diabetic coma medication history physical disease onset history of low blood sugar suddenly laboratory sweating, palpitation, character abnormalities, coma, insulin, glibenclamide and other dilated pupils, fast heartbeat, sweating, blurred consciousness, coma, blood glucose <2.8 mmol / L (50mg/dl) urinary glucose (-) 1-24h ketoacidosis, polyuria, thirst, nausea, vomiting, abdominal pain, no medication history or stop insulin glucose breath with mild to moderate dehydration Kusmaul 16.7-33.3mmol / L , urine sugar, urine ketone strong positive non-ketotic hyperosmolar diabetic coma 1-14d old age, dehydration, 40% can be no history of diabetes diuretics, hormones, dialysis, severe dehydration, blood pressure, blood glucose has pathologic reflex 33.3mmol / L, urine positive, ketone (-) or (


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