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Condition analysis:
Hello! You this consideration is vertigo.
Suggestion:
Can take Sibelium stop treatment and dizziness, the other one must pay attention to the diet to restore. Vertigo patients nutritious diet should be based on the principle of light and fresh. To eat eggs, meat, vegetables and fruit. Bogey Feigan spicy food items, such as fatty meat, fried things, wine, and pepper. Second, to carry out the spirit nursed back to health. the spirit of vertigo patients nursed back to health can not be ignored. depression, anger and other mental stimulation can cause hyperactivity or liver wind stirring, and induced vertigo. Therefore, the vertigo patients should be broad-minded spirit of optimism, ease of mind, emotional stability, which is to prevent vertigo attacks and reduce the number of attacks is very important. Third, pay attention to rest living. overtired or Lack of sleep is one of the factors inducing vertigo. regardless of when or after the onset of vertigo should pay attention to rest. in the acute attack of vertigo should stay in bed. vertigo patients to ensure adequate sleep is important. in sufficient sleep, its symptoms reduce or disappear.on the answer: a total of 3, to paragraph 10 Peng 5,259,660 to support the answer: good answer, I'll add it!
syuna1989: good answer, I'll add it!
syuna1989: Thank you,

Benign paroxysmal positional vertigo induced by postural changes of the main symptoms of vertigo as a cl


inical syndrome. The syndrome, lesions in the vestibule and the cochlear canal, 50 to 70 years the incidence in the elderly as the main object of young uncommon.
Clinical features: the first bit in a particular induced transient dizziness, with nystagmus. Typical performance for the incidence of sudden, intense rotatory vertigo occurs, often disappear after a few seconds or tens of seconds, rarely more than 1 minute. The vertigo is usually turned to lie down in the seat, or by lying to a sitting position, or when turning over in bed, mostly to one side of vertigo, but vertigo greater degree of change, the head severe dizziness minor activities that attack.
There are many conditions can cause benign paroxysmal positional vertigo, for example, otolith membrane spontaneous transformation, the labyrinth concussion, otitis media, ear surgery and the anterior branch of the vestibular artery obstruction, may become triggers of vertigo; especially the ear produced within the semicircular canal in the small otoliths, the elderly often lead to the most common cause of vertigo. The small stones can be the first place or position with the rotation of the scroll in the ear, touch your ear cilia, the imbalance of the brain to convey information to cause dizziness and other symptoms. Otolith vertigo caused by rolling are common, but easily misdiagnosed. Have a general understanding of the disease, symptomatic and treatment.
Treatment: The main use of reduction therapy. Specialists according to the patients by the specific circumstances of head position and posture by rotation, so that semicircular canal rolling stones, the stones into the oval balloon inside the ear. After reset, the vast majority of patients symptoms completely disappeared, usually without medication. Long duration of individual dizziness, reset ineffective in patients with short-term application of brain and nourishing can hydrochlorothiazide, flunarizine, promethazine and other anti-symptomatic treatment of vertigo Yaowu to alleviate the symptoms. Han Yongxia
Etiology and symptoms have been confirmed in the posterior semicircular canal cristae within the basophilic granular material. Ridge sediments (Shen stone crest syndrome) may be derived from the otolith of a calcium carbonate crystals, normal implantation balloon in the inner ear and the oval capsule plaque. utricular otolith membrane spontaneous transformation, lost concussion, otitis media, ear surgery and the anterior branch of the vestibular artery occlusion, which may be related to the etiology and pathogenesis factors. benign paroxysmal positional vertigo in patients with ear to one ear or the other side, or take the lead when tilted back to the point of . nystagmus may also occur, but not associated with hearing loss or tinnitus. benign paroxysmal positional vertigo can be subsided within a few weeks to months. but may in a few months or years after the recurrence. diagnosis of positional nystagmus can be a challenge test. were sitting on the examination table, and then let the head turned to one side, and immediately taken to lower the head down in the supine position at one end of the examination table. latency of several seconds can occur after severe vertigo, which lasted about 15 ~ 20 seconds, accompanied by rotary nystagmus. as affected ear is the ear, when the head turned to the left, nystagmus clockwise; as affected ear is the right ear, nystagmus was counterclockwise. When the patient re-take seats, the reaction repeated attacks, but the nystagmus when lying above the opposite direction, the degree of light. reaction was fatigue properties, it is immediately repeated the test in the next, the reaction will subside. In the end, or central nervous system lesions can occur in the positional nystagmus. latency from the reaction and subjective feeling of the severity of fatigue, limited Response duration and direction of rotation nystagmus, benign paroxysmal positional vertigo of the central nervous system disorders conducted from the difference. the location of the central nervous system diseases, no nystagmus latency, fatigue and severe subjective feeling, and as long as position to maintain, the aforementioned reaction can continue. nystagmus due to the central nervous system diseases, which can be vertical or direction change type, if it is rotating, then it may be wrong to type (that is, with the expected opposite direction.) diagnostic evaluation should include hearing tests, cold tests ENG and gadolinium-enhanced MRI, with particular attention to the internal auditory canal in order to rule out other conditions such as acoustic neuroma. treatment tell patients to avoid inspire position. such as benign paroxysmal positional vertigo disease has reached 1 year, usually by the tympanic pathway, isolated and suffering from ear cut off the supply of the posterior semicircular canal nerves were eased. In some cases, this treatment can lead to hearing damage. Variable name Variable name positional vertigo ampullary crest otolith disorder (Cupulalithiasis), more common in adults aged 40 to 60, women more common. Harrison (1975) reported 365 cases, 60% of unknown etiology. Barany was first reported in 1921, 1 case of 27-year-old female patient, head to the right side of the dizziness symptoms, that is the performance of utricular lesions. Schuknecht (1962) of 3 cases of pathological utricle, saccule and ampulla crest sensory epithelium of normal, only the posterior semicircular canal cristae are calm basophilic granules, may be sensitive to the incentives to stimulate production of gravity, it is known as the ampulla crest otolith disease. Diagnosis 1. Cephalic nystagmus examination so that patients sit on the bed, first sit-nutation position, observed 10 seconds after no vertigo and nystagmus, so sit up and then observed for 10 seconds, and then make the first lateral side sit, observe 10 seconds then sit nutation to the other party to observe 10 seconds, each change in position, sit and lie down should be completed within 3 seconds, such as when in a position nystagmus, should continue to observe 30 seconds, such as nystagmus continued does not disappear is the test positive. Such as the right ear down to the right when the rotary nystagmus, eye gaze to the left of the vertical nystagmus appeared, repeated tests were positive, known as non-fatigue-type; the other hand, no longer appears repeatedly nystagmus test, known as fatigue-type . Head position in a different direction nystagmus appeared unchanged as directed type; if there nystagmus in different directions are called variable to type. Where a single head position in nystagmus occurs, short duration, has an incubation period, directional type of fatigue responders, mostly peripheral lesions; otherwise mostly central lesions. As tracking and optokinetic tests, and other generally normal. 2. Hot and cold temperature such as vestibular function tests normal. 3. Pure tone hearing test were normal. Treatment (a) of the conservative treatment of this disease prognosis is good, about a year died in the self-healing. Long-term healing of the individual who can be symptomatic therapy, such as oral and stability, vulgar cellulose drugs. Exercise can also be used to force posture, the use of a fatigue response characteristics of position on the day fixed in the induced long-term exercise, long time to adapt to no longer have a sense of vertigo. Such as severe dizziness, application of 4% lidocaine, do intratympanic streptomycin sulfate, to be received good effect. (B) of the surgical treatment of long-term conservative treatment fails, may take the vestibular neurectomy treatment, conditions are best to do after the ampullary nerve transection, can simply remove the posterior semicircular canal cristae cure the abnormal impulses. Ear stapes surgery with incisions, skin grafts will be turned to the front of the tympanic membrane, exposed hammer, anvil joints and the round window, round window with a diamond drill will be rubbed off the bottom level of 1 ~ 2mm. In order to facilitate surgical operation, after the first drill to the external auditory canal wall under the bone rubbed off a little, to make clear the round window exposed. Ampullary nerve after appropriate Reattachment in the round window membrane within the inner edge of 1mm, discern the nerve under a microscope, with the angle, and cut off from the will of its complex back to the tympanic membrane, filling the external auditory canal. Pathology 1. May be due to spontaneous degeneration of the utricle, otolith detached from the capsule plaque deposition in the post-ampullary crest. 2. Inflammation or trauma can also induce this disease, such as viral labyrinthitis or chronic otitis media process, white blood cells, phagocytes, or quality of the dander into the endolymph; brain trauma or stapes surgery, the blood into the lymph solution, can form a visible particles, and deposited in the ampullary crest. Barber reported that longitudinal temporal bone fractures, 47% of the patients may present positional vertigo, no fracture occurred were also 20% of this levy. Dix and Hallpike (1952) reported 100 cases of otitis media patients, 26% and a positional vertigo. Ear surgery such as tympanoplasty, mastoidectomy vestibular and other damage can also be complicated by the disease. 3.Lindsay, Cawthorne pathologic findings, the former Pretrial artery and the vestibular nerve branches to the utricle and the superior semicircular canal and the lateral semicircular canal degeneration, while the balloon and the posterior semicircular canal were normal, suggesting that the high blood pressure, heart disease, and vertebral artery insufficiency , can cause arterial embolization prior to pre-trial that may cause degeneration of otolith membrane, shedding, deposition in the ampullary crest. hypergammaglobulinemia, immunoglobulin deposition in the cristae, or alcoholics alcohol to thin cristae, can produce the sign. Children with severe hearing loss to wear the high-strength (120 ~ 130dB) hearing aids, about half of the occurrence of positional vertigo. Once the formation of cristae stone, take the lead, take an upright position, the posterior semicircular canal is vertical crest, like the side in the affected ear, the posterior semicircular canal cristae be converted into horizontal position, calm after the crest has an ear, due to gravity deviation of the role of the ampulla can cause irritation and vertigo and nystagmus occurred. Particles, such as inflammatory adhesion is not strong, several transformation position, the particles fall off stray, no longer constitutes a stimulus, then fatigue phenomenon. Conversely, if otolith adhesion strong, durable non off, despite repeated change position, the stimulus intensity diminished, may constitute a non-fatigue model, which is constant in unhealed, can affect the life and work, the need for surgery. Clinical manifestations 1. The whole body in good condition, only a position or head position when dizziness, nausea, vomiting, such as changing position to the other side, a good quick improvement in symptoms and repeated after a certain position there. General incubation period 2 to 3 seconds, less than 1 minute duration, with little nausea, vomiting, cold sweats phenomenon, less occurrence of dumping. 2. No vertigo, tinnitus, deafness phenomenon, has not increased cochlear symptoms. 3. The disease more than a few weeks or months in the mitigation expert, individuals can also be adjourned to a few years. 4. Sick period without headaches and no other central nervous signs. And the following differential diagnosis should be differentiated from various diseases. (A) of the central position of the fourth ventricle of vertigo is common in brain tumors and vascular lesions, may have headache, nausea, vomiting, and other positive neurological signs. Often spontaneous nystagmus, postural nystagmus test in 30 seconds or more sustained, no latency, no fatigue, CT and MRI can be further confirmed. (B) the sudden onset of vestibular neuritis, for days, hot and cold test reduced, there are upper respiratory tract infection, mild increase in blood leukocytes. (C) of the cervical spine bone hyperplasia, may compress the vertebral artery or cervical sympathetic stimulation caused by vertebral artery spasm, the vestibular insufficiency, more common in adults over 40 years of age, vertigo and specific location of the head and neck. Often associated with other vertebral artery insufficiency symptoms such as headache, visual impairment and upper limb numbness. Cervical spine X-ray shows the first 4 to 6 cervical spine bone hyperplasia or other abnormalities.


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