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Home treatment of symptoms induced by causes of disease prevention and check the article Symptoms of vasomotor rhinitis
Almost everyone has occasional nasal symptoms so different from the normal nose and nasal disease is sometimes difficult but as long as double-check carefully detailed history of nasal symptoms induced by factors that total more than 1 hour per day course for up to one month following the exclusion of more than illness can be diagnosed as vasomotor rhinitis 1. allergic rhinitis allergen skin test positive eosinophils in nasal secretions and basophils were seasonal onset of seasonal rhinitis 2. infection with acute rhinitis and chronic rhinitis rhinitis and nasal discharge often divided mucus or mucopurulent secretions in the mostly neutrophils 3. very allergic vasomotor rhinitis addicted . the symptoms: All the content vasomotor rhinitis screening method
1. Before rhinoscopy 2. An X ray picture clear and complications of nasal sinus infection. Vasomotor rhinitis inspection methods: the entire contents of the cause of vasomotor rhinitis
Frequently-occurring disease as predisposing factor in the disease mechanism for the treatment of complex integrated measures should be taken to (a) avoid or remove the triggers to improve working conditions and environment do not control the pace of life and stable emotional fatigue and stress on the necessary psychological treatment for patients with or suggestive language had a noticeable effect sometimes cau


sed by endocrine factors, as the case should be to help treat your endocrinologist (b) changes in drug therapy should be considered in due course the disease the drug of choice 1. nasal decongestants on the nasal symptoms can be used as the main but applications to the attention of the occurrence of drug-induced rhinitis may take intermittent or alternating administration of the three P . the cause of vasomotor rhinitis: all on disease-related vasomotor rhinitis article SSI ļ ʱ

Vasomotor rhinitis is nasal blood vessels of neuroendocrine gland functional regulation of imbalance caused by a high nasal reactivity. Pathogenesis of this disease complex, many aspects remain unclear, it is a definite clinical diagnosis and effective treatment with to be difficult. no significant gender differences in the incidence of children with vasomotor rhinitis rarely occur.According to risk factors, Goldman will be divided into three vasomotor rhinitis clinical types, this classification for the diagnosis and treatment of some significance.
First, the physical nature of reactive
Some patients often stimulated for some reason specific nasal reactions. If each in with cold air, sudden temperature changes, moisture, etc., that produce paroxysmal sneezing, nasal discharge accompanied by more water. Patients often can be clearly stated predisposing factors. This type of reverse may also be too strong rhinitis.
Second, the mental reaction type
Mental stress, fear or resentment, frustration and other repeated stimulation, causing nasal response in patients. Modern neural changes in the content and pace of life accelerated the conflict between traditional values and so increase the number of such patients.This type of patient often can not find any suspicious predisposing factor, accounting for vasomotor rhinitis the majority of cases. Patients are often water nasal discharge, nasal edema, polyps or nasal mucosa from time to time. Endocrine dysfunction may be the incentive of one. (Related: The best ENT Hospital Shanghai)
TAG Tag: What are the symptoms of rhinitis what is the cause of rhinitis
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Vasomotor rhinitis (vasomotor rhinitis) is a neuroendocrine on nasal blood vessels, glandular imbalance caused by functional regulation of a highly reactive nasal. The complex pathology of the disease, many aspects remain unclear, it is a definite clinical diagnosis and effective treatment has brought some difficulties. No significant gender differences in the incidence of children with vasomotor rhinitis rarely occur. Etiology and pathology
Pathogenesis
Contains a lot of nasal glands, rich of many of the vascular bed and the nerve to form a fine, sensitive and active end organ, the exercise of its respiratory portal of various physiological functions. It relies on nerve - blood vessels, nerves - endocrine and other activities, maintain the balance of the nasal cavity and the external environment. This balance depends on the surface of the two pathways from the hypothalamus: one by the pituitary gland function by means of the implementation of the endocrine chain humoral regulation of the nasal mucosa, one directly through the autonomic nervous system, the implementation of the neural regulation. If one of these pathways function changes occur, can cause nasal blood vessels, glandular dysfunction, increased reactivity, vasomotor rhinitis which is a major pathophysiological basis of disease.
(A) of the autonomic nervous system dysfunction
Under normal circumstances, sympathetic nerve activity, its peripheral release of norepinephrine and neuropeptide y, by the corresponding receptors in the vessel wall to maintain vascular tone in the nasal mucosa. Parasympathetic stimulation, its peripheral release of acetylcholine, and the glands in the vascular wall through the M receptor to cause vasodilation and glandular secretion. Recent study has also found there in the nasal parasympathetic in vasoactive intestinal peptide (VIP) immunoreactive fibers. Pterygoid canal nerve stimulation (main with parasympathetic), the VIP immunoreactive fibers release VIP, which can cause blood vessels to dilate, and this vasodilation can not be blocked by atropine. Uddman (1987) that the excitement caused by the parasympathetic glandular secretion is due to acetylcholine, and vasodilation is derived from the non-cholinergic vasodilator-like factor - VIP.
Repeated sympathetic stimulation (fatigue, irritability, anxiety, stress, etc.), can consume excessive amounts of neurotransmitter synthesis enzymes and have been synthesized and reserve transmitters in the nerve endings, so that 1 and receptors in a corresponding reduction in the number of can cause reduction of sympathetic tone. Some anti-hypertension drug, non-selective -blockers and antidepressants are sympathetic blockers, repeated applications can result in decreased sympathetic tone. Decreases in sympathetic tone, increased parasympathetic nervous excitability, can cause vasodilation, glandular secretion of strong, resulting from vasomotor rhinitis symptoms. As early as 1943, Fowler discovered that allows resection of cervical sympathetic ganglia in patients with vasomotor rhinitis. Removal of superior cervical sympathetic postganglionic animals can lead to nasal blood vessels to dilate, submucosal edema, glandular hyperplasia, and found increased activity around the gland cholinesterase. These changes in patients with vasomotor rhinitis mucosa pathological findings are identical. [Food in China]
(B) endocrine disorders
Endocrine disorders can also cause changes in nasal reactivity. Hypothyroidism can cause reduced autonomic nervous sympathetic tone. These patients often the main symptoms of nasal obstruction. Changes in estrogen levels may also cause nasal symptoms. Many clinical data prove that some female patients in the premenstrual or significant nasal symptoms during pregnancy, such as nasal congestion, multiple sneezing, clear tears. Animal experiments confirmed that the animal can increase the body estradiol levels significantly increased nasal reactivity, showing that nasal epithelial thickening, edema, expansion of small blood vessels, glands hyperplasia. This mechanism of action of estradiol is not clear. However, increased levels of estradiol have been found when the body can make nasal tissue increased cholinergic M-receptors, 1-adrenergic receptor decreased. Estradiol also enhanced the release of mast cells in non-immune role of histamine.
(C) histamine and other inflammatory mediated
Quality of non-immune causes the release of non-immune release of histamine and other media, a variety of factors, such as chemical (anesthetic, salicylic acid preparations), physical (sudden changes of temperature, humidity, weather, dust), nerve of (emotional changes) and so on. Histamine release of non-specific immune mechanism is not clear, but regardless of exactly how the mechanism is governed by the regulation of intracellular cAMP levels. As long as the level of intracellular cAMP decreased, so that mast cells can release media.
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