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(Wuchuan City Chinese Medicine Hospital, Guangdong 524500)
Hemifacial spasm is a difficult disease clinic, one of the from July 1995 ~ end of 1998, the use of high-frequency electroacupuncture for treatment of 26 patients with hemifacial spasm, and achieved good effect. Are reported below.
1 General Information
A total of 26 patients in this group, are in out-patients, 9 males and 17 females; the youngest 29 years old, maximum 68 years; the shortest duration of 1 month, maximum of 4 years. 16 cases of eye twitching, facial twitching and numbness of the mouth in 9 cases, jaw and ear after the Ministry of scalp twitch in 1 case.(1) Main points: Ashi point, that first appeared in the local spasm or severe spasm of the site; (2) Distribution Point: eye twitch or four white with the sun, face and mouth twitch with zygomatic or in warehouses, jaw and ear After the scalp


or cheek twitching with Yifeng car.
2.2 How
Each treatment will take the main points, with points to take a, 2 points for a group. Routine disinfection, rapid flat thorn (acupuncture direction and the local direction of the facial nerve to go try to keep the same line) was followed by G 6805 gas electric acupuncture treatment for bipolar, choose a continuous wave, frequency, take the largest scale, the current intensity slowly increased from 0 to facial muscle spasm was sustained contraction (note: not beating), but also the degree of patient can tolerate. Full Text: 981 words Price: 0.20 You are not logged, please login / register
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Abstract hemifacial spasm that face the side of the seizure (convulsions individuals have bilateral), the spirit of the more intense, more serious emotional spasm. Hemifacial spasm as the initial symptoms of eyelid twitching, people have "left eye jump Choi, right eye jump disaster," said, it is generally not cause people's attention, after a period of lesion formation and development of a hemifacial spasm, and even Scroll to the mouth, severe joint and neck. Hemifacial spasm can be divided into two types, one of the original hair hemifacial spasm, one is the sequelae of facial paralysis generated hemifacial spasm. Two types can be distinguished from the symptoms on it. Of the original hair hemifacial spasm, in the stationary state may also occur, cramps relief after a few minutes, without control; facial paralysis sequelae of hemifacial spasm, only to do blink, brow lift and other activities generated.
Hemifacial spasm (Hemifacial Spasm, HFS) is a half side of paroxysmal involuntary muscle twitching, usually limited to one side of the face, which is also called hemifacial spasm, even visible on both sides. Began more than in the orbicularis muscle, and gradually to the cheek and the whole hemi-facial development, the development of rare reverse. May be due to fatigue, tension and intensified, especially in speech, smiling significantly, and in severe spasticity can be presented. Onset in middle age more than the minimum coverage for the two years of age. In the past a good fat that women in recent years, statistics show that the incidence of gender. HSF development to the end, a few mild cases of facial paralysis can occur.
, vascular factors such as reported in 1875 Schulitze case of HFS patients undergoing autopsy and found that the presence of facial parts, "cherry" size of the basilar artery. Currently known about 80% to 90% of the HFS is due to the facial nerve vascular compression out of existence due to brainstem area. Clinical data show that: in the vascular factors lead to HFS to anterior inferior cerebellar artery (AICA) and posterior inferior cerebellar artery (PICA)-based, while the superior cerebellar artery SCA) second. SCA is known from the bottom of the basilar artery and posterior cerebral artery junction, take the line of the most constant, while the relative variation of PICA and the AICA is large, thus easy to form a loop or ectopic vascular compression of the facial nerve; lost another variation on the arteries and other arteries such as the vertebral artery, basilar artery formation of facial nerve compression may result in HFS. Previously thought: HFS is due to the pulsatile arterial pressure due to recent years, studies have shown that: a single vein surface of nerve compression can lead to HFS, and the blood vessels can form two or more persons jointly facial nerve compression, which to some extent HFS on the impact on surgical outcome.
, non-vascular factors in the cerebellar pontine angle (CPA) of the non-vascular lesions such as granulomas, tumors and cysts and other factors can also produce HFS. The reason may be due to: placeholder lead normal vascular displacement. Singh et al reported a case of CPA epidermoid cyst in the AICA compression of the facial nerve led to the shift HFS; occupying the direct compression of facial nerve; occupying the impact of abnormal blood vessels themselves, such as arteriovenous malformations, meningioma, aneurysm, etc. Also some of the posterior fossa lesions can also lead to HFS. If the middle of a rare nerve Schwann cell tumor of facial nerve compression caused by HFS. Hirano reported a case of cerebellar hematoma in patients whose initial symptoms HFS. In younger patients, the local HFS arachnoid thickening may be one of the main produce, and some congenital diseases such as Arnold-Chiari malformation and congenital arachnoid cysts can occur even HFS.
, other factors in the brain stem areas of facial nerve compression factor is the existence of the main causes of HFS, and most of the scholars during the cerebellar pontine angle surgery observed: facial nerve out there areas beyond the brain stem vascular compression does not produce HFS. And Kuroki, etc. observed in animal models: the facial nerve from the brainstem in areas beyond the facial nerve demyelination, which can be expressed as EMG changes similar to HFS. Mar-tinelli also reported a case of facial nerve injury can occur around the support HFS. Brain stem area on the facial nerve out there areas other than the compression factor is causing the HFS, still need further study.
In addition, HFS is also seen in some systemic diseases such as multiple sclerosis. HFS only a few familial cases reported so far, the mechanism is not clear, presumably related to heredity.
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